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7, 8, 9 collectively, these observations have. Adapted from gidding ss, champagne ma, de ferranti sd, et al. Thereafter, helen hobbs and jonathan cohen found that individuals with loss of function in pcsk9 identified in the dallas heart study seemed to enjoy protection from atherosclerotic cardiovascular.

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Apo (b) = apolipoprotein (b) Pcsk9 = proprotein convertase subtilisin/kexin type 9 Hefh = heterozygous familial hypercholesterolemia

Hofh = homozygous familial hypercholesterolemia